Natural killer cell-mediated cytotoxicity, JAK-STAT signaling pathway, small cell lung cancer, chemokine signaling pathway, B-cell receptor signaling pathway, acute myeloid leukemia, apoptosis, cytokine, and cytokine receptor interaction, and nonsmall cell lung cancer were significantly enriched in the EDAR high-expressed phenotype (Figure 5b). This evidence concerns the gene SOAT1 and lung cancer.