GSEA revealed that the high-risk group was predominantly participated in autoimmune disease, cytokine-cytokine receptor interaction, ECM receptor interaction, JAK-STAT signaling pathway, natural killer cell-mediated cytotoxicity, and neuroactive ligand receptor interaction (Figure 11F), while low-risk group was primarily regulated by the pathways associated with TCA cycle, DNA replication, oxidative phosphorylation, pentose phosphate pathway, pyrimidine and pyruvate metabolism, and RNA degradation (Figure 11G). This evidence concerns the gene SOAT1 and autoimmune disease.