Given that both HNF1A and HNF1B motifs were enriched in mSWI/SNF-dependent sites and both HNF1 family transcription factors mediated viral infection, yet only HNF1A was shown to bind BAF complexes, we performed reciprocal immunoprecipitations to determine whether HNF1A/B factors dimerized. This evidence concerns the gene HNF1B and viral infectious disease.