Whereas mice lacking or mutant for Adamts10 have not been reported to develop WMS1-like lens defects including microspherophakia, ectopia lentis, and cataract (Mularczyk et al. 2018; Wang et al. 2019), homozygous Em/J mice developed cataract with full penetrance—suggesting that Adamts10 is a marginal candidate gene for Em. Here, ADAMTS10 is linked to erythema multiforme.