They found that CD63-and major histocompatibility complex (MHC) II-associated EVs collected from the serum of DMD patients were significantly enriched in myomiRs compared to healthy controls, especially miR-1, miR-133a, and miR-206 for CD63-associated EVs and miR-1 and miR-133a for MHC II-associated EVs. The gene discussed is CD63; the disease is Duchenne muscular dystrophy.