IL10 and infection: The comparable lung bacterial burden (Fig 1E) among the two mouse groups at the time of analysis: i) excludes the possibility that the observed discrepancy in IL-10 production between the WT and μMT mice is the result of different numbers of M. tuberculosis in the lungs of infected animals; and ii) suggests that the augmented lung expression of the immunosuppressive IL-10 (Fig 4) in the tuberculous B cell-deficient mice does not compromise TB control in these animals in the chronic phase of infection in the model studied.