SCN5A and Arrhythmia: Since calcium transients were employed to show a phenotype feature (arrhythmia), and their changes are indirectly related to loss‐of‐function of SCN5A channels, and more importantly, the mechanisms underlying the abnormal calcium transients observed in BrS‐hiPSC‐CMs are unclear, the subsequent experiments with respect to fever and LPS effects on BrS features focused on alterations of SCN5A channels and possible mechanisms for the alterations.