CP and COVID-19: Thus, CP IgA, but not CP IgG, was considered a putative marker of chronic infection and subsequent risk of death from not only ischemic heart disease by rupture of atheromatous plaques but also multiple organ dysfunction by endothelial injury. Similarities in the inflammatory processes operating in COVID-19 and atherosclerosis have been suggested [6], and it is possible that arteriosclerosis and endothelial injury due to past CP infection affect the prognosis of COVID-19.