In conclusion, as shown in Fig. 10, baicalin magnesium has a therapeutic effect on HFD-induced NASH rats, which may act by reducing lipid deposition and oxidative stress, down-regulating the expression of NLRP3 inflammatory vesicles, and inhibiting the expression of caspase-1 and IL-18. This evidence concerns the gene NLRP3 and metabolic dysfunction-associated steatohepatitis.