In support of this notion, other studies also shows that tumor cell‐derived, cDC1‐dependent T cell infiltration contributes to antitumor immunity in vivo.[41, 42] We speculated that RA might induce a component shift of MoDCs (CD14+ CD206+) and pDCs (CD11− B220+) to cDC1s (CD103+ CD11+ DCs) in TME. This evidence concerns the gene ITGAE and neoplasm.