In murine models, after exposure to pesticides such as TBT, mancozeb, imidacloprid, lambda-cyhalothrin, fipronil, dicamba, and oxadiazon, an increase in PPARα and/or PPARγ receptor exposure is shown in adipose tissue composition, the presence of dyslipidemia (elevated lipids in the blood) and an increase in lipid metabolism associated with an increase in the concentration of ROS and inflammatory biomarkers in adipose tissue, liver, and muscle [80]. Here, PPARA is linked to metabolic syndrome.