Additionally, there is accumulating evidence that the T-helper 2 (Th2) inflammatory response phenotype can induce protective effects against the COVID-19 immunopathogenesis due to the increased secretion and release of Th2 anti-inflammatory cytokines such as IL10, IL4, and IL13 and recruitment of the eosinophils to the site of inflammation (159). The gene discussed is IL4; the disease is COVID-19.