The anti-α-smooth muscle actin (αSMA) antibody stained perivascular smooth muscle cells densely and smooth muscle or myofibroblasts in some stroma less densely (Figure S3), which possibly reflected the density and expression of αSMA (e.g., cancer-associated fibroblasts [CAFs], which differentiate into cells with the myofibroblast phenotype, are morphologically similar to smooth muscle cells but have variable αSMA expression depending on the degree of differentiation). Here, ACTA1 is linked to cancer.