Importantly, it should be noted that mutations in another member of the SLC2 family, i.e., SLC2A1 (encoding the glucose transporter type 1), are associated with a variety of distinct epilepsy syndromes, e.g., early onset absence epilepsy, childhood absence epilepsy, and myoclonic-atonic epilepsy (Hildebrand et al., 2014; Koch and Weber, 2019). Here, SLC2A1 is linked to epilepsy.