The systemic consequences of high concentrations of adipocytokines, interleukins, small dense LDL particles, and nonesterified fatty acids and the reduced clearance of ApoB, triglycerides, and very low‐density lipids are related to impaired liver function, increased insulin resistance, glucose intolerance, and procoagulation factors [2]. The gene discussed is APOB; the disease is Glucose intolerance.