Here, we discovered that, senescent IPF cells treated with nintedanib presented elevated levels of Bcl-2 when compared to normal senescent cells, despite previous studies showing nintedanib inhibits apoptotic proteins in lung-resident myofibroblasts35, although the effect was not evaluated in senescent cells, which are naturally resistant to apoptosis36. The gene discussed is BCL2; the disease is idiopathic pulmonary fibrosis.