They observed that the proportion of mannose receptor (MR)+ MDC type 1 in patients with endometriosis was increased, and the expression of IL-1β and IL-6 was elevated in the co-incubation system, indicating that MR+ DCs enhanced the phagocytotic ability of dESCs and stimulated inflammation, contributing to the pathogenesis of endometriosis (96). Here, NR3C2 is linked to endometriosis.