In a mouse model treated with Zearalenone (ZEA), a Fusarium-produced toxin that acts like estrogen, the inhibition of estrogen signaling activities decreased the number of neutrophils and the expression of pro-inflammatory cytokines by suppressing the NF-κB signaling pathway, which inhibits the growth of endometriosis (109). Here, NFKB1 is linked to endometriosis.