IL-17A triggered the expression of angiogenic factors, pro-inflammatory cytokines, and chemotactic cytokines, including granulocyte colony-stimulating factor (G-CSF), CXCL12, CXCL1, and CX3CL1, which play a crucial role in promoting the establishment and growth of endometriosis lesions (111). This evidence concerns the gene IL17A and endometriosis.