ANGPTL7 and neoplasm: Tumor-derived blood vessel morphogenesis genes and host-derived neutrophil degranulation gene sets were depleted in Angptl7-knockdown conditions, while core matrisome (tumor), RNA translation (host), and TCA transport (host) persisted or increased in Angptl7-knockdown condition compared with nontargeting control (SI Appendix, Fig. S7C).