A previous study on a mouse model overexpressing CELF1 and developing DCM (Wang et al, 2015), identified down‐regulation of Transcription factor A mitochondrial (Tfam), Apelin (Apln), and Long‐chain fatty acid‐CoA ligase 1 (Acsl1) as potentially associated with DCM. The gene discussed is APLN; the disease is familial dilated cardiomyopathy.