Cisplatin (Pt) binds to DNA to form a complex that inhibits DNA cleavage by deoxyribonucleases[12] and cooperates with NK cell‐induced pyroptosis to promote the release of Pt‐DNA complexes in tumor cells and activate the cyclic GMP‐AMP synthase‐stimulator of interferon genes (cGAS‐STING) pathway in antigen‐presenting cells (APCs), leading to substantial interferon secretion.[13] Interferon reverses the immunosuppressive tumor microenvironment, further potentiating NK cell activation. Here, CGAS is linked to neoplasm.