Reports have shown that ICH‐induced mice neurological deficits, memory impairment, and brain atrophy are reduced by Fer‐1 treatment,28 and the administration of Fer‐1 upregulates Fpn, decreases the iron content, and then improves the lipid peroxidation and early brain injury in subarachnoid hemorrhage rats.29 The gene discussed is SLC40A1; the disease is subarachnoid hemorrhage.