Several studies report these associations with COVID-19 due to evidence, such as that of the specific inhibition of the NLRP3 inflammasome, which alleviated excessive lung inflammation and COVID-19-like pathology in ACE2 transgenic mice infected with SARS-CoV-2 [23], and that the fact that inflammasomes designated as NLRP3 were active in the peripheral blood mononuclear cells (PBMCs) and serum samples of COVID-19 patients as well as in the postmortem lung tissues of patients who had passed away [24]. Here, NLRP3 is linked to COVID-19.