Furthermore, ARDS caused by COVID-19 differed from non-COVID-19 ARDS in that the former generated an increased expression of CCL5/RANTES while reducing IL-2, TNF-related apoptosis-inducing ligand (TRAIL), and granulocyte-colony-stimulating factor (G-CSF) [61]. This evidence concerns the gene TNFSF10 and acute respiratory distress syndrome.