To determine if a decrease in interstitial inflammation and alveolitis seen on day nine in immunosuppressed hMPV-infected animals treated with MPV467 was associated with altered pulmonary cytokine/chemokine expression, levels of pulmonary MIP-1α and IP-10 (mediators linked to lung injury and immune dysfunction [37,38,39,40]) were measured. Here, CCL3 is linked to hypersensitivity pneumonitis.