NRXN1 and autism: To dissect the genetic complexity of autism at the NRXN1 locus, we have collected and generated an allelic series of mouse models carrying distinct CNVs across the Nrxn1 locus, including ΔExon1, that has been studied previously but not systematically for ΔExon1 heterozygotes, and two new mouse models: the first bearing a deletion of Nrxn1α exon 9 that disrupts Neurexin 1α protein translation (named as ΔExon9 hereafter), and the second carrying an ASD proband-associated ~20kb deletion in intron 17 of Nrxn1α, upstream of the Nrxn1β promoter (named as ΔIntron17 hereafter).