On the contrary, this study reveals that Cx36 deletion increases the incidence at the postnatal period of CSB that later disappears during posterior maturation, as occurs in the preterm and term infants with CSB (Fenner et al., 1973), indicating that the high level of Cx36 expression during the first weeks of life (Belluardo et al., 2000; Solomon, 2003) protects against CSB. The gene discussed is GJD2; the disease is Cockayne syndrome type 2.