TNF and pulmonary fibrosis: Amiodarone and COVID-19 share similar pathophysiology toward the development of pulmonary fibrosis through the upregulation of the renin-angiotensin-aldosterone system (RAAS) and cytokines (interleukin (IL)-1, IL-8, tumor necrosis factor (TNF)-α, and transforming growth factor (TGF)-β) [4,5].