Due to elevated levels of the proinflammatory molecules, the increased cytokines and chemokines have been seen in COVID-19 patients, such as interleukin (IL)-1ß, IL-2, IL-6, IL-17, tumor necrosis factor (TNF-α), interferons (IFN-γ), and chemokines (CXCL8 and CXCL10), which play a pivotal role in both pathogenesis of GBS and the rapid progression of GBS associated with COVID-19 (Fig. 1). This evidence concerns the gene CXCL10 and COVID-19.