In subsequent immunohistochemical stainings, the tumor cells exhibited a protein expression profile compatible with a smooth muscle cell-like differentiation with a diffuse and strong immunoreactivity for vimentin, a patchy but strong immunoreactivity for alpha-smooth muscle actin (a-SMA) and a negative immunoreactivity for desmin, as well as pan-cytokeratin. This evidence concerns the gene SMN1 and neoplasm.