Several studies have already been published concerning genistein treatment during cervical cancer development, and the reported activities of genistein include inducing cell cycle G2-M phase arrest and apoptosis, inhibiting the action of histone deacetylases and DNA methyltransferases, and synergizing the radiation effect by cell cycle G2-M phase arrest and AKT (Protein Kinase B) activation [21,22,23,24,25,26]. This evidence concerns the gene AKT1 and cervical cancer.