Although it is difficult to draw conclusions on the impact of virus coinfection on a possible miRNA-mediated fibrosis, it is interesting to note that we previously demonstrated that HCMV/HHV-6A simultaneous infection increased TNFα and other fibrosis-associated factors and that miR-19b and miR-155, both induced by coinfection, were reported to trigger endothelial IL-6 and TNFα production [68]. This evidence concerns the gene TNF and infection.