Although the mechanisms have not been yet established in glioblastoma, neurons express mir23a/b, and mir23a controls EMT in GBM by targeting homeobox D10 (HOXD10) [290], whereas mir23b is downregulated by Myc in ischemic neurons, leading to nuclear factor erythroid 2-related factor 2 (Nrf2) upregulation and reductions in apoptosis and the infarcted area [291]. Here, NFE2L2 is linked to glioblastoma.