NLRP3 and lupus nephritis: Experimental data reported by studies performed using different models of kidney injury, such as chronic glomerulonephritis [37,38,39,40], diabetic nephropathy [41,42], Lupus Nephritis [43], crystalline nephropathy [44,45,46,47] and hypertensive nephropathy [48,49,50], have shown that NLRP3 inflammasome activation participates in the underlying pathogenetic events involved in the onset and progression of kidney damage, regardless of the aetiology of kidney disease.