ALB and myotonic dystrophy type 1: The impact of vorinostat on a DM1 mouse model overexpressing 250 CTG repeats, driven by the human skeletal actin promoter (HSALR), was next assessed; a colony of wild-type mice (WT) and mice expressing HSA containing non-pathogenic CTG repeat lengths (less than 50) (HSASR) were used as non-diseased baseline models.