The activations of TLR4/MYD88 and ULK1/ATG7 signals, which decrease the expression of miR18* and miR4802, respectively, are among the proven mechanisms of action of this bacterium, important both in enhancing CRC cell proliferation, mechanisms of CRC chemo-resistance (5-fluorouracil (5-FU) and oxaliplatin) and in cell autophagy. This evidence concerns the gene TLR4 and colorectal carcinoma.