Doxorubicin-induced senescence promoted the recruitment of Treg cells and myeloid-derived suppressor cells to mediate apoptotic resistance in diffuse large B-cell lymphoma (DLBCL) via pro-inflammatory cytokines, such as IL-2, IL-6, IL-8, IL-10, IL-35, TGFβ, and VEGF [77]. The gene discussed is IL2; the disease is diffuse large B-cell lymphoma.