Ventricular tissues from rabbit models displayed increased levels of DRP1 and no variation in MFNs [145]; a different result was obtained in an in vivo model of catecholaminergic polymorphic ventricular tachycardia (CPVT) aggravated by the mutation in the trans-2, 3-enoyl-CoA reductase-like (TECRL) gene which encodes for an endoplasmic reticulum protein, published to be associated to inherited arrhythmia [146,147]. Here, DNM1L is linked to catecholaminergic polymorphic ventricular tachycardia.