GLUD1 and glioblastoma: As we have shown that IL-11Rα allows for glutaminolysis, glutaminolysis-dependent survival, and reduced apoptosis in glucose-starved conditions, and as we observed that inhibition of GLUD1 by EGCG and R162 and GSS by BSO (two key enzymes of glutaminolysis) led to the reversal of this survival, we next determined if IL-11Rα expression correlated with GLUD1 and GSS expression in glioblastoma patient samples.