In contrast, AL amyloidosis (p = 0.0588), tubulointerstitial nephritis (p > 0.9999), diabetic kidney disease (p = 0.2425), IgA vasculitis (p > 0.9999), monoclonal dense deposit disease (DDD, p = 0.4730), post-infectious glomerulonephritis (GN, p > 0.9999), and thrombotic microangiopathy (p > 0.9999) presented CRP elevation that was not significantly different from that of renal vasculitis (Figure 1A). This evidence concerns the gene CRP and dense deposit disease.