Table 3 shows key molecules that positively or negatively affect both the angiogenic and decidualization processes in adenomyosis. Among them, at least the HIF1A [51] and SLIT2 [52] genes potentially impair endometrial decidualization and receptivity. As detailed in the next section, overexpression of the HIF gene, which is most important for angiogenesis, has been reported to reduce endometrial receptivity in experimental animal models via suppression of HOXA10 and HOXA11 [51]. The gene discussed is HOXA11; the disease is adenomyosis.