CSNK2B and Poirier-Bienvenu neurodevelopmental syndrome: The authors suggested that the pathogenic variants affecting p.Asp32 lead to upregulation of CSNK2B expression at the transcript and protein level, along with global dysregulation of canonical Wnt signalling, and through a dominant negative effect, causing a new intellectual disability-craniodigital syndrome (IDCS) distinct from POBINDS [14].