Although both the IHC and Western blot assays confirmed that SRSF2, SRSF7, HNRNPF, and HNRNPR were upregulated in AA PCa vs. EA PCa (Figure 6B and Figure S6), only the SRSF2- and HNRNPF-binding motifs were identified within intron 19 (a flanking intron upstream of PIK3CD) by using the SFmap program [39]. The gene discussed is HNRNPF; the disease is posterior cortical atrophy.