Recently, other preclinical studies of cervical cancer have recommended inducing E-cadherin expression by stimulating the expression of miR-185-5p [201], using miR-126 for the induction of cytotoxicity mediated by TNF-α and FasL [202] and using lncRNA HOX (HOTAIR—long non-coding RNA HOX transcript antisense), which acts competitively against miR-148a and has demonstrated an ability to participate in the regulation of HLA-G expression [203]. Here, CDH1 is linked to cervical cancer.