In patients with acute myeloid leukemia (AML), the presence of internal tandem duplication (ITD) in the Fms-like tyrosine kinase (FLT3) gene has been associated with similar induction success as wild-type FLT3, but much shorter remission duration, higher likelihood of early relapse and shorter overall survival (OS) [1,2,3]. This evidence concerns the gene FLT3 and acute myeloid leukemia.