Other predictive factors, including nuclear expression of BCL10 and NF-κB [22], NK cell infiltration, expression of CD86 [23], a large proportion of CD19- and CD20-positive cells [24], high expression of cMET [25], high microsatellite instability (MSI) [26], and high values of Hp and CagA antibodies [27], contribute to poor responsiveness to eradication therapy in gastric MALT lymphoma. This evidence concerns the gene HP and MALT lymphoma.