Specifically, Elbehairi et al. reported that, in PCa cells, the Pd(II) complex of Gboxin analog-chitooligosaccharides conjugate (Pd(II)COS@GbA) effectively impairs ATP synthase expression and activity, leading to the suppression of ATP production, and triggers mitochondrial fragmentation (fission) by increasing the expression levels of the fission protein DRP1 while decreasing those of the fusion protein OPA1 [262]. Here, DNM1L is linked to posterior cortical atrophy.