Considering the functional relevance of this 3′UTR variant, which has been previously associated with vitiligo [19] as well as several pathological conditions [63,64,65,66,67,68,69], and whose role in the HLA-G mRNA stability has been well established in vitro [63], we reinforce that this SNP is likely to be the causal variant directly associated with vitiligo susceptibility. The gene discussed is HLA-G; the disease is vitiligo.