Perini et al. [31] analyzed NK cells before and after interferon beta administration and found that the drug acts on the depletion of CD3-CD16+ and CD3-CD57+ NK cells, while Al-Falahi et al. [25], in animal models with experimental autoimmune encephalomyelitis, showed that glatiramer-acetate has a much stronger killing efficiency of one’s own NK cells against dendritic cells. This evidence concerns the gene IFNB1 and experimental autoimmune encephalomyelitis.