Excessive oxidative stress and abnormal production of proinflammatory cytokines are associated with colitis pathogenesis [17,18,19] with the aberrant release of proinflammatory cytokines, including TNF-α, IFN-γ, IL-6, IL-8, IL-12, and IL-17 [20,21,22]. Here, CXCL8 is linked to colitis.