The onset and progression of PCa can be prevented by modifying eating habits through modulation of the nuclear factor kappa B (NF-κB), target of rapamycin in mammals (mTOR), mitogen-activated protein kinase (MAPK), Akt, extracellular signaling regulated kinase (ERK), and phosphoinositide 3-kinase (PI3K) signaling pathways. Here, AKT1 is linked to posterior cortical atrophy.