SOD1 and metabolic dysfunction-associated steatotic liver disease: The lncRNA RMRP directly regulated miR-206 and inhibited its activity on PTPN1, which aggravated NAFLD; therefore, Yin et al. demonstrated that RMRP inhibition in rats with NAFLD delayed the progression of the disease, significantly reduced MDA accumulation, and boosted the antioxidant defenses, as shown by increased SOD activity [112].